全部

1·Objective:to explore the way to cure keloid.

目的：探讨治疗瘢痕疙瘩的方法。

2·Objective To identify the genetic alteration in human keloid.

目的了解瘢痕疙瘩遗传学改变的特征。

3·Conclusion Survivin might play a role in the development of keloid.

结论生存素可能参与瘢痕疙瘩的发病。

4·Methods We divided keloid into inertia type and active type clinically.

方法：我们临床上将瘢痕疙瘩分为惰性类和活跃类。

5·Conclusion: Keloid can be effectively treated with triamcinolone acetonide.

结论：曲安奈德注射液能够有效治疗瘢痕疙瘩。

6·Methods: keloid fibroblasts were isolated from fresh keloid tissue and cultured.

方法取新鲜瘢痕疙瘩组织，体外培养瘢痕成纤维细胞。

7·Methods The ultrastructure of keloid was observed by transmission electron microscope.

方法用透射电镜观察瘢痕疙瘩的超徽结构。

8·Results The patients, 47 cases were treated 147 keloid, All incisions primarily healed.

结果：本组病例47例，共治疗147个瘢痕疙瘩，切口均甲级愈合。

9·Conclusion P53 can obviously inhibit MMP1 and MMP3 expression in human keloid fibroblasts.

结论P 53蛋白能够抑制人瘢痕疙瘩成纤维细胞MMP1、MMP3蛋白的表达。

10·Purpose To clarify the molecular pathological basis of infiltrating growth and onset of keloid.

目的：探讨瘢痕瘤的发生及其侵袭性生长失控的分子生物学基础。