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1·This new paradigm of oncogenesis has been validated in a growing list of tumors.

这一新的癌变模式已在越来越多的肿瘤中得到证实。

2·The results provided data for the study on HPV virological oncogenesis of the oral Cancers.

这一结果为开展口腔癌的HPV病因学研究提供了参考资料。

3·Conclusion Telomere reduction and telomerase activation are critical factor during oncogenesis.

结论端粒缩短并激活端粒酶是涎腺肿瘤形成的重要因素。

4·The instability of genome which results from defect of MMR genes is thought as a new oncogenesis way.

由于错配修复基因缺陷而导致的基因组不稳定，被认为是导致肿瘤发生的一条新途径。

5·PML is a growth and transformation suppressor, and it may play a role in the oncogenesis of solid tumors.

PML是一个生长和转录抑制因子，研究表明PML表达变化可能和一些实体癌的发生有关。

6·Although there are many HPV serotypes, two of them - 16 and 18 - account for the lion's share of the oncogenesis.

尽管有很多HPV血清型，其中两种—16和18—在引发肿瘤中占绝大多数。

7·Substantial evidence shows that inflammation promotes oncogenesis and, occasionally, participates in cancer rejection.

充实的证据表明炎症能够促进肿瘤发生，偶尔也会参与癌症排斥。

8·Activation of telomerase is the critical step in cellular immortality and oncogenesis. Its activity is controled by many factors.

端粒酶激活是细胞永生化和肿瘤形成的关键步骤，其活性受到多种因素的调控。

9·If one is really, for instance, captivated by the biochemistry of protein kinases, you don't necessarily have to work on kinases in oncogenesis.

比如说，如果你真正着迷于蛋白激酶生物化学，你不一定要在肿瘤学范围内研究激酶。

10·Telomerase activation is essential for the stabilization of chromosomes in proliferation cells, leading to cellular immortality and oncogenesis.

端粒酶激活可以维持增殖细胞染色体的稳定性，导致细胞永生化和肿瘤发生。