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1·Objective to explore the role of chemokines in experimental autoimmune neuritis (EAN).

目的探讨趋化因子在实验性自身免疫性神经炎(EAN)中的作用。

2·Chemokines and chemokine receptors are important regulatory factors in tumor behaviors.

趋化因子及其受体是肿瘤细胞生物学行为的重要调节因素。

3·Objective To analyze the types of chemokines expressed in murine medullary thymic dendritic cell line (MTDC).

目的分析小鼠胸腺树突状细胞株(MTDC)中化学趋化因子的类型。

4·In recent years, it is suggested that chemokines can promote tumor cells proliferation, migration, and invasion.

近年来，有研究证实趋化因子能促进肿瘤细胞增殖、侵袭、迁移。

5·Conclusion Upregulated chemokines were associated with macrophage recruitment and destruction of islet xenografts.

结论：趋化因子的上调与巨噬细胞的聚集、胰岛移植物的破坏有关。

6·The researchers found that astrocytes in mice were producing chemokines in response to activation of their TNFR molecules.

研究人员发现，小鼠的星形胶质细胞可以产生化学增活素作为对TNFR激活以后的应答。

7·Therefore, the new theory and the technology about chemokines may provide some new ways for clinical treatment of certain diseases.

因此，有关趋化性细胞因子的新理论和技术可为临床治疗某些疾病提供了新思路。

8·Conclusion Cetirizine may exert the anti-allergic inflammatory effects of skin by inhibiting the expression of chemokines in human keratinocytes.

结论西替利嗪可能通过抑制角质形成细胞趋化因子的表达而发挥抗皮肤过敏炎症作用。

9·When TNFR is activated, it causes cells to send out signal molecules called chemokines that direct immune cells to the site of damage or infection.

当TNFR被激活以后，它使细胞释放出信号分子-化学增活素，化学增活素引导免疫细胞到达破坏或感染的位点。

10·In addition to inflammation, platelets stimulate repair by releasing growth factors and chemokines to recruit circulating vascular progenitor cells.

除了炎症反应外，血小板通过释放生长因子和趋化因子刺激修复得以募集循环中血管内皮组细胞。