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1·Conclusions Anti-pain powder by oral administration combined with peripheral opioid receptors could ha.

目的证明口服抗痛粉是否有强镇痛效应。

2·In this review, the distribution and physiologic functions of endogenous opioid peptides in hypothalamus were discussed.

本文主要对内阿片肽在下丘脑中的分布特点及生理功能作了介绍。

3·The research team discovered changes in mu opioid receptors and dopamine transporters of the brain -- areas important to reward pathways.

研究团队发现在的末梢神经及大脑的多巴胺传输中，区域对酬谢路径非常重要。

4·Every day I see variations at both the beginning and end of life: a young man abandoned by friends as he struggles with opioid addiction;

每一天，我都会在生命的起点和终点看到这类情况的种种版本：一个年轻的男人在竭力对抗毒瘾之际遭到友人的抛弃；

5·It is suggested that the endogenous opioid peptidergic system might act as an important modulatory system between CNS and the immune system.

提示中枢内阿片肽系统参与创伤应激介导的免疫功能低下，而电针具有调整创伤应激介导的免疫机能低下效应的作用。

6·These results are interesting because we saw changes in opioid receptor gene expression in a brain area that controls how much we eat during a meal, said Bello.

贝洛克说，这些结果很有趣，因为我们看到随着脑部阿片受体基因的改变，它控制着我们一顿饭吃多少。

7·It appears to involve more primitive aspects of the brain, activating deep structures that may block pain at a spinal level — similar to how opioid analgesics work.

这似乎涉及到大脑更原始的问题，激活深层结构可能会阻止在脊椎级的疼痛-与阿司匹林片类镇痛药的作用相似。

8·The invention employs the side effects of the opioid formulation to permit patients to self-limit drug intake, thereby avoiding toxicity while achieving analgesia.

本发明利用了该阿片样物质制剂的副作用使得患者可以自我限制药物摄入，从而在达到止痛效果的同时避免了中毒。

9·Especially, some of them show biphasic modulation on opioid functions, which enhance opioid analgesia, but inhibit tolerance to and substance dependence on opioids.

特别是有些药物能对阿片功能产生双向调节作用，即增强阿片镇痛，对抗阿片耐受和躯体依赖。

10·Conclusion: Melatonin can change the responses of pain neurons to pain stimulation via opioid receptor in the LHb, which might be one of analgesic mechanisms by MEL.

结论：褪黑色素可通过作用于外侧缰核的阿片受体而影响其痛相关神经元对痛刺激的反应，这可能是褪黑色素中枢镇痛机制之一。