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1·Therefore, deficiency of costimulatory molecules on tumor cells is one of the important mechanisms of tumor immune escape.

可见，肿瘤细胞上共刺激分子缺陷是肿瘤免疫逃逸的重要机制之一。

2·It has been showed that the expressions of costimulatory molecules are associated with xero organ transplantation rejection.

已有研究表明，共刺激分子的表达与同种异体移植排斥反应有关。

3·TLR2 is reported to enhance the expression of costimulatory molecules on the antigen presenting cell, however the regulatory mechanisms are still unclear.

已知tlr 2活化会促进抗原呈现细胞表面共同刺激分子的表现，但其调控机制及讯息传递路径仍不是非常清楚。

4·CONCLUSIONS: The whole blood gene expression quantities of costimulatory molecules CD154 and ICOS reasonably robustly differentiated rejection patients from control patients.

结论：共刺激分子CD 154和ICOS的全血基因表达量能可靠的区分排异患者和对照患者。

5·Objective: to explore the expression characteristics of intracellular cytokines and surface costimulatory molecules in embryo antigen tolerant t cells transferred in pregnant mice.

目的：分析过继转输的小鼠胚胎抗原耐受T细胞内细胞因子及细胞表面协同刺激分子的表达特征。

6·Objective to explore the significance of costimulatory molecule B7-1 (CD80) and MHC molecules in the pathogenesis of hematologic malignant tumors and in the immuno-genetic therapy.

目的探讨共刺激分子B7 1 (CD80)在血液系统恶性肿瘤发病学及免疫基因治疗中的重要作用。