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1·Different degrees of myofibril damage were found.

肌原纤维也有不同程度损伤。

2·Meantime the property of the myofibril contraction under different biochemistry conditions had been tested.

同时，对各种生化条件下肌纤维的收缩性质进行了测试。

3·Injuries of capillaries promoted the formation of contraction band (CB) of myofibril and myocardial necrosis.

毛细血管损伤促进了肌原纤维收缩带形成，加速了心肌细胞坏死。

4·The inhibition of Myostatin expression can accelerate the myoblast proliferation and myofibril hypertrophy, which results in the skeletal muscle hyperplasia.

对该基因表达的抑制可引起成肌细胞的过度增殖和肌纤维的肥大，从而引起骨骼肌增生。

5·The high content (11.93%) of Lys residue distinguished carp MBSP from other serine proteinases and this may account for its myofibril-binding characteristic.

MBSP有高含量的赖氨酸残基（11.93%），此特性可能与该酶的肌原纤维结合特性有关。

6·CONCLUSION: Myocardial interstitial fibrosis and the reduction of myofibril elements are the major pathological basis for the impairment of cardiac function in patients with DCM.

结论：DCM患者心肌间质纤维化，肌原纤维减少是扩张型心肌病心功能损害的主要病理基础。

7·The effect of soybean trypsin inhibitor (STI) on the degradation of silver carp myofibrillar proteins caused by an endogenous myofibril-bound serine proteinase (MBSP) was studied.

研究了大豆胰蛋白酶抑制剂（STI）对鲢肌肉中肌原纤维结合型丝氨酸蛋白酶（MBSP）引起的肌原纤维蛋白降解作用的影响。